How does the Oxford Recovery Model help with Strokes?

There are no drugs on the market that can restore brain cells which have been damaged due to stroke. And while various kinds of therapies can be helpful, they cannot address the root cause of underlying brain imbalances.

So, what are the root causes of the imbalances created by a stroke? Recent advances in neuroscience reveal the root causes include a lack of blood flow and oxygen to the brain, lack of specific brain chemicals, and brainwave imbalances.

Let’s look at all these causes a little more in depth: Inside the brain, we have billions of brain cells, called neurons, sending electrical signals to each other, which we measure as brainwaves. These electrical signals cause the brain cells to release small chemicals called neurotransmitters, such as serotonin, dopamine, and adrenaline.

This vast network of brain cells is fed with oxygen and other nutrients by blood from over 100,000 miles of blood vessels inside the brain. For people who have experienced a stroke, there is often not enough of these blood vessels supplying oxygen and important nutrients.

This lack of nutrients begins to cause brainwave imbalances, as measured by an EEG scan. This, in turn, causes a downstream effect on brain chemicals called neurotransmitters, meaning that the brain cells start producing too little or too much of the chemicals they need. Neurotransmitters imbalances are further exacerbated by nutritional deficiencies in the person’s diet, especially of key vitamins, minerals, antioxidants, and amino acids.

So, how do we start correcting a potentially multifaceted problem?

One of our most important tools at Oxford Recovery Center is called hyperbaric oxygen. This therapy actually causes new blood vessel growth in the brain and causes the brain to generate new stem cells which turn into new brain cells. (See our video about hyperbaric oxygen here.)

Another important tool is neurofeedback therapy. We use neurofeedback to scan your brain’s electrical signals and reset them with non-invasive feedback in the form of sound. (See our video about neurofeedback here.)

And finally, neurotransmitter therapy and dietary coaching is used to correct nutritional deficiencies which are underlying the brain chemical imbalances—imbalances that we can find through laboratory testing we provide.

We strive to offer the most comprehensive research-based recovery program available for stroke victims, and we are happy to serve you by taking as much time as we need to answer your specific questions.

Research Studies

Click on the links below to discover more about the science behind the Oxford Recover Model’s approach

• [Clinical study on acupuncture combined with hyperbaric oxygenation for improving balance function of cerebral infarction]
• [Hyperbaric oxygen improves long-term learning-memory deficits and brain injury in neonatal rat with hypoxia-ischemia brain damage]
• A Review of Oxygen Therapy in Ischemic Stroke
• Confirmation of medical benefits of Oxygen therapy during a Stroke
• Delayed HBOT is more effective than early prolonged normobaric hyperoxia in experimental
• Effect of large dose hyperbaric oxygenation therapy on prognosis and oxidative stress of acute permanent cerebral ischemic stroke in rats
• HBOT attenuated the decrease in regional glucose metabolism of rats subjected to focal cerebral ischemia – a high resolution positron emission tomography study
• Hyperbaric Oxygen Reduces Tissue Hypoxia and Hypoxia-Inducible Factor-1alpha Expression in Focal Cerebral Ischemia
• Hyperoxia preconditioning – the next frontier in neurology?oxfordhbot.com/…ibrary/stroke/270.008.pdf
• Neuroprotection by Oxygen in Acute Transient Focal Cerebral Ischemia Is Dose Dependent and Shows Superiority of HBOT
• Preconditioning with HBOT attenuates brain edema after experimental intracerebral hemorrhage
• Proliferation of neural stem cells correlates with Wnt-3 protein in hypoxic-ischemic neonate rats after HBOT
• Protection of mitochondrial function and improvement in cognitive recovery in rats treated with HBOT following lateral fluid-percussion injury
• The Effect of Oxygen Therapy on Brain Damage and Cerebral pO(2) in Transient Focal Ischemia in the Rat