TBIs (Traumatic Brain Injuries)
Hope for a Better Life after TBI
An integrative approach, combining multiple therapies, delivers the best results for TBI Recovery.
TBI Therapies Offered In our Michigan Healing Centers
Types of Brain Injury
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Anoxic Brain Injury
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Hypoxic Brain Injury
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Traumatic Brain Injury
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Concussion
Past Clients Experienced the Following Benefits from Our TBI Program
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Clearer speech
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Increased mobility
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Better memory, focus, and concentration
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Improved mental function and clarity
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Stabilizes mood changes and irregularities
Click the video below to watch one of our past clients experience at our facility after his TBI!
Video Script
Rick, how has oxford recovery center affected your life? “Oxford Recovery Center has restored the quality in my life, which had been gone for many years.” What was your life like before you came here? “Dark, withdrawn, not sociable, my family, even my wife, said that she didn’t like me for eight and a half years and now it’s totally different.” Rick, what happened that brought you here and how long ago was that? “I had my second back surgery in March of this year, and I finally got permission to drive I was driving through town and I had seen your sign for years but didn’t know what it meant. I thought I’d pull in and so I met with, I forget who it was, and they showed me your brochure and I looked at the one side and I thought ‘boy this could really help, perhaps speed up my recovery’ and then I looked at the other side and I noticed the TBI and the anxiety, depression and all the other things – PTSD – and all the other things that I had been dealing with for over eight years.” So wait let me get this straight, you came in here because you were concerned about your back, “correct”, but you had a whole another issue that you didn’t even know we could help. ”You’re right.” So how long ago did you have, I know you got shot, how long ago was that? “That was the December the 11th, 2007 at 1:30 in the afternoon.” So you were shot in the head, “right”, basically left for dead. “Correct.” And for all of these years nobody has given you any hope. “No, none whatsoever. The PTSD group, the private groups, the VA, psychiatric care, the brain life center, I didn’t feel like I got any results from that. My wife says that she saw a difference but I didn’t feel it and neither did anybody else. But this has just changed my life entirely. Everybody I know has just- they can’t believe that this is me, and it’s the old me.” Well I find it as one of my best Christmas gifts to see rick again, coming in, and telling me that he got his life back. So thank you for sharing and Merry Christmas. “Merry Christmas to you and everybody here, you’ve all been such wonderful people and I can’t say enough good about you, I really can’t. I’ll be a friend for life.” Merry Christmas. “Merry Christmas”
Therapies at Oxford Recovery Center
Click on the links below to learn about the therapies offered at Oxford Recovery Center
Hyperbaric Oxygen Therapy (HBOT)
Physical Therapy and Suit Therapy
QEEG/Neurofeedback with Real-time Brain Imaging
Nutrition and Weight-Loss
Research and News
For more information about the research and news behind hyperbaric and TBI please click on the links belowResearch
- [Hyperbaric oxygen improves long-term learning-memory deficits and brain injury in neonatal rat with hypoxia-ischemia brain damage]: Researchers found that HBOT significantly improves long-term learning-memory deficits and attenuates brain injury in rats with hypoxia-ischemia brain damage [Article in Chinese]
- A prospective, randomized clinical trial to compare the effect of hyperbaric to normobaric hyperoxia on cerebral metabilism, intracranial pressure, and oxygen toxicity in severe traumatic brain injury: Researchers found that HBOT produces a brain tissue P02 greater than or equal to 200 mm Hg more robustly than normobaric hyperoxia (NBH).
- Delayed hyperbaric oxygenation is more effective than early prolonged normobaric hyperoxia in experimental focal cerebral ischemia: Researchers concluded that HBOT is more effective than normobaric oxygen (NBO) therapy in transient experimental ischemia.
- Effect of hyperbaric oxygen administered at different pressures and different exposure time on differentiation of neural stem cells in vitro: Researchers found that HBOT at the 2 ATA and 60 minutes treatment protocol produced the best effect in the differentiation of neural stem cells (NSCs) into stem cells in vitro [Article in Chinese]
- Hyperbaric oxygen therapy (1.5 ATA) in treating Sports Related TBI/CTE: two case reports: Dr. Kenneth Stoller, MD identifies that HBOT may be adopted as a non-invasive therapy to treat acute and chronic traumatic brain injury.
- Hyperbaric oxygen treatment attenuated the decrease in regional glucose metabolism of rats subjected to focal cerebral ischemia – a high resolution positron emission tomography study: Researchers found that early exposure to HBOT can partially reverse the downward trend for glucose utilization in ischemic areas, which could be responsible for reported beneficial effects of HBOT for permanent cerebral ischemia.
- Hyperbaric oxygen therapy for traumatic brain injury: Dr. Huang and Dr. Obenaus found HBOT is neuroprotective and exerts more robust and long-lasting effects in the absence of pulmonary or cerebral oxygen toxicity than normobaric oxygen therapy (NBOT). Additionally, findings show that HBOT shows neuroprotective effects in the absence of incrased oxygen toxicity at pressures less than 3 ATA.
- Hyperbaric oxygen induces endogenous neural stem cells to proliferate and differentiate in hypoxic-ischemic brain damage in neonatal rats
- Hyperbaric oxygen therapy promotes neurogenesis: Where do we stand?
- Hyperbaric oxygen in traumatic brain injury
- Hyperbaric oxygen preconditioning induces tolerance against brain ischemia–reperfusion injury by upregulation of antioxidant enzymes in rats
- Hyperbaric oxygen therapy improves spatial learning and memory in a rat model of chronic traumatic brain injury
- Neuroprotective effects of hyperbaric oxygen treatment on traumatic brain injury in the rat
- Pilot case study of the therapeutic potential of hyperbaric oxygen therapy on chronic brain injury
- Preconditioning with hyperbaric oxygen attenuates brain edema after experimental intracerebral hemorrhage
- Pressure-related Increase of Asymmetric Dimethylarginine Caused by Hyperbaric Oxygen in the Rat Brain: A Possible Neuroprotective Mechanism
- Proliferation of neural stem cells correlates with Wnt-3 protein in hypoxic-ischemic neonate rats after hyperbaric oxygen therapy
- Protection of mitochondrial function and improvement in cognitive recovery in rats treated with hyperbaric oxygen following lateral fluid-percussion injury
- The effect of oxygen therapy on brain damage and cerebral pO(2) in transient focal ischemia in the rat
- Treatment of mild traumatic brain injury with hyperbaric oxygen
- Hyperbaric Oxygen Therapy Can Improve Post Concussion Syndrome Years after Mild Traumatic Brain Injury – Randomized Prospective Trial
- CD34+/CD45-dim stem cell mobilization by hyperbaric oxygen – changes with oxygen dosage “Because hyperbaric oxygen treatment mobilizes bone marrow derived-stem/progenitor cells by a free radical mediated mechanism, we hypothesized that there may be differences in mobilization efficiency based on exposure to different oxygen partial pressures. Blood from twenty consecutive patients was obtained before and after the 1st, 10th and 20th treatment at two clinical centers using protocols involving exposures to oxygen at either 2.0 or 2.5 atmospheres absolute (ATA). Post-treatment values of CD34+, CD45-dim leukocytes were always 2-fold greater than the pre-treatment values for both protocols. Values for those treated at 2.5 ATA were significantly greater than the 2.0 ATA treatment group by factors of 1.9 to 3-fold after the 10th and before and after the 20th treatments. Intracellular content of hypoxia inducible factors -1,-2, and -3, thioredoxin-1 and poly-ADP-ribose polymerase assessed in permeabilized CD34+ cells with fluorophore-conjugated antibodies were twice as high in all post- versus pre-treatment samples with no significant differences between 2.0 and 2.5 ATA protocols. We conclude that putative progenitor cell mobilization is higher with 2.5 versus 2.0 ATA treatments, and all newly mobilized cells exhibit higher concentrations of an array of regulatory proteins.”
News
- Brain injury sufferer recovers 90%
- Car Accident Victim Waking up with HBOT
- Drew Christy’s Recovery
- HBOT Study on TBI Recovery
- Hyperbaric Oxygen Therapy Provides a wide number of benefits for Traumatic Brain Sufferers
- Neurologist Exposes the Benefits of HBOT for TBI and PTSD
- Representatives of the International Hyperbaric Medical Association Testify Before House Veterans Affairs Committee
- Retired NFL player finds hope for his brain in oxygen chamber
- Successfully Treating Brain-Injured Veterens
- US Defense Department Looking at HBOT for treatment of TBI
- US Soldiers with TBI and PTSD to be evaluated with HBOT
- Positive Influence of HBOT on Recovery from Brain Trauma
Interested in Oxford’s Unique Approach to Healing?
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